ATLANTA - A mutated version of a common cold virus has caused 10 deaths in the last 18 months, U.S. health officials said Thursday. Adenoviruses usually cause respiratory infections that aren't considered lethal. But a new variant has caused at least 140 illnesses in New York, Oregon, Washington and Texas, according to a report issued Thursday by the U.S. Centers for Disease Control and Prevention.
CDC officials don't consider the mutation to be a cause for alarm for most people, and they're not recommending any new precautions for the general public.
"It's an uncommon infection," said Dr. Larry Anderson, a CDC epidemiologist.
The illness made headlines in Texas earlier this year, when a so-called boot camp flu sickened hundreds at Lackland Air Force Base in San Antonio. The most serious cases were blamed on the emerging virus and one 19-year-old trainee died.
"What really got people's attention is these are healthy young adults landing in the hospital and, in some cases, the ICU," said Dr. John Su, an infectious diseases investigator with the CDC.
There are more than 50 distinct types of adenoviruses tied to human illnesses. They are one cause of the common cold, and also trigger pneumonia and bronchitis. Severe illnesses are more likely in people with weaker immune systems.
Some adenoviruses have also been blamed for gastroenteritis, conjunctivitis and cystitis.
There are no good antiviral medications for adenoviruses. Patients usually are treated with aspirin, liquids and bed rest.
Some people who get infected by the new bug probably would not suffer symptoms, and some may just feel a common cold. Sick people should see a doctor if they suffer a high fever or have trouble breathing, Anderson said.
In the CDC report, the earliest case of the mutated virus was found in an infant girl in New York City, who died last year. The child seemed healthy right after birth, but then became dehydrated and lost appetite. She died 12 days after she was born.
Tests found that she been infected with a form of adenovirus, called Ad14, but with some little differences, Su said.
It's not clear how the changes made it more lethal, said Linda Gooding, an Emory University researcher who specializes in adenoviruses.
Earlier this year, hundreds of trainees at Lackland became ill with respiratory infections. Tests showed a variety of adenoviruses in the trainees, but at least 106 — and probably more — had the mutated form of Ad14, including five who ended up in an intensive care unit
In April, Oregon health officials learned of a cluster of cases at a Portland-area hospital. They ultimately counted 31 cases, including seven who died with severe pneumonia. The next month, Washington state officials reported four hospitalized patients had the same mutated virus. One, who also had AIDS, died.
The Ad14 form of adenovirus was first identified in 1955. In 1969, it was blamed for a rash of illnesses in military recruits stationed in Europe, but it's been detected rarely since then. But it seems to growing more common.
The strain accounted for 6 percent of adenovirus samples collected in 22 medical facilities in 2006, while none was seen the previous two years, according to a study published this month in the medical journal Clinical Infectious Diseases.
The new bug could have implications for the military. Other forms of adenoviruses have been a common cause of illness in recruits. Military officials are bringing back an adenovirus vaccine — administered as a pill — that was given to recruits from 1971 to 1999, CDC officials said.
A Barr Pharmaceuticals vaccine for the military, currently being tested, is expected to be licensed in 2009. Like the old pill, it focuses on adenovirus serotypes 4 and 7, because those bugs have been persistent problems, said Col. Art Brown, an Army physician involved in the product's development.
Some CDC officials said a vaccination against the mutant Ad14 might be needed. Brown said it isn't clear if the mutant Ad14 will be an enduring threat, but the military will monitor illness reports.
"If it persists, then we'd consider if the vaccine needs to be modified further," said Brown, of the U.S. Army Medical Materiel Development Activity.
2007年11月19日星期一
2007年11月17日星期六
Musculoskeletal Regeneration Research Receives First-Of-A Knd ' Emerging Frontiers' NSF Grant
Musculoskeletal Regeneration Research Receives First-Of-A Knd ' Emerging Frontiers' NSF GrantA research group led by Dr. Cato T. Laurencin, with faculty representing five departments at the University of Virginia, will work on a first-of-its kind, $2 million grant project as they explore novel methods for the regeneration of musculoskeletal tissues. The grant from the National Science Foundation is known as an EFRI grant -- Emerging Frontiers in Research and Innovation. The group, led by Dr. Laurencin, with faculty in orthopaedic surgery, chemical engineering, biomedical engineering, electrical engineering and materials science, will investigate several innovative ways to engineer new material surfaces that will allow a range of musculoskeletal tissues to grow. "These studies should give us important fundamental information that will be broadly applicable in tissue engineering and regenerative medicine," said Dr. Laurencin, principal investigator (PI) for the studies and Professor of Orthopaedic Surgery, Biomedical Engineering and Chemical Engineering at the University of Virginia. "This grant complements my recently awarded Department of Defense grant aimed at exploring new strategies for limb regeneration." Laurencin said that a team approach is needed to tackle such a great problem. "The National Science Foundation Grant allows for a broad team that will explore important material surface cues to permit optimum cellular interactions. Learning how best to design materials, create artificial tissues and understand their healing abilities -- ultimately, for the successful treatment of our patients -- is what this translational research program aims to do," Laurencin said.
2007年11月16日星期五
Musculoskeletal Regeneration Research Receives First-Of-A Knd ' Emerging Frontiers' NSF Grant
Musculoskeletal Regeneration Research Receives First-Of-A Knd ' Emerging Frontiers' NSF GrantA research group led by Dr. Cato T. Laurencin, with faculty representing five departments at the University of Virginia, will work on a first-of-its kind, $2 million grant project as they explore novel methods for the regeneration of musculoskeletal tissues. The grant from the National Science Foundation is known as an EFRI grant -- Emerging Frontiers in Research and Innovation. The group, led by Dr. Laurencin, with faculty in orthopaedic surgery, chemical engineering, biomedical engineering, electrical engineering and materials science, will investigate several innovative ways to engineer new material surfaces that will allow a range of musculoskeletal tissues to grow. "These studies should give us important fundamental information that will be broadly applicable in tissue engineering and regenerative medicine," said Dr. Laurencin, principal investigator (PI) for the studies and Professor of Orthopaedic Surgery, Biomedical Engineering and Chemical Engineering at the University of Virginia. "This grant complements my recently awarded Department of Defense grant aimed at exploring new strategies for limb regeneration." Laurencin said that a team approach is needed to tackle such a great problem. "The National Science Foundation Grant allows for a broad team that will explore important material surface cues to permit optimum cellular interactions. Learning how best to design materials, create artificial tissues and understand their healing abilities -- ultimately, for the successful treatment of our patients -- is what this translational research program aims to do," Laurencin said.
2007年11月14日星期三
Diesel Air Pollution Linked To Heart Attack And Stroke In Healthy Men
Diesel Air Pollution Linked To Heart Attack And Stroke In Healthy MenUK and Swedish researchers found that diesel fumes from road vehicles increased blood clots and platelets in healthy volunteers. These are symptoms closely linked to increased risk of heart attack and stroke.The researchers reported the results of a small study to a meeting of the American Heart Association's Scientific Sessions 2007 held in Orlando, Florida, earlier this week.Previous observational and epidemiological studies have also shown a close link between exposure to traffic pollution and heart attack, said study lead author Dr Andrew Lucking, who is a cardiology fellow at the University of Edinburgh in Scotland, UK."This study shows that when a person is exposed to relatively high levels of diesel exhaust for a short time, the blood is more likely to clot. This could lead to a blocked vessel resulting in heart attack or stroke," said Lucking.Lucking and colleague carried out a double blind, randomized cross-over study on 20 healthy male participatns aged from 21 to 44. Using a specially designed exposure chamber, the men were separately exposed to filtered air (this was the control) and then to 300 mg per cubic metre (mcg/m3) of diesel exhaust fumes, which is roughly the concentration you breathe in while standing by a busy street.The researchers measured clot formation, blood coagulation, platelet activity and markers of inflammation by attaching each participant to a perfusion chamber and allowing a small amount of blood to pass through it. This was done 2 hours after exposure and then again 6 hours after exposure.Clot formation was assessed by passing the blood through a special shear chamber that simulates the types of pressure the blood would be under in blood vessels. The researchers tested the blood at high shear and low shear.Platelet activation was assessed by measuring the number of platelets associated with white blood cells. When platelets are activated they stick to white blood cells like neutrophils and monocytes and form clumps, thereby playing a key role in the formation of blood clots.The results showed that: Breathing diesel fumes increased clot formation in the low shear chamber by 24.2 per cent compared to breathing filtered air.In the high shear chamber the increase in clot formation from diesel fumes was 19.1 per cent.These effects were observed at both 2 and 6 hours after exposure to diesel fumes.Breathing diesel fumes increased platelet-neutrophil aggregates from 6.5 to 9.2 per cent 2 hours after exposure.It also increased platelet-monocyte aggregates from 21 per cent to 25 per cent 2 hours after exposure.But at 6 hours after exposure the platelet activation increases due to diesel fumes were not statistically significant.Lucking said: "High levels of traffic pollution are known to increase the risk of heart attack in the immediate hours or days after exposure."He said this study showed a "potential mechanism that could link exposure to traffic-derived air pollution with acute heart attack."Although these results apply to diesel engine fumes, it's not clear whether gasoline powered engines would have the same effect, said the researchers. Diesel fumes contain a much higher concentration of very fine particles, they said.Diesel engines are on the rise because they offer superior fuel economy, but, as Lucking explained:"While diesel engines burn more efficiently, they also put more fine particulate matter into the air."The researchers said while exercise was good for people with cardiovascular disease, they would not recommend they exercise near traffic congestion.The UK and Swedish team will be working together on the next step, which is to test the effectiveness of the particle traps fitted to diesel engines to reduce exhaust particles."Exposure to air pollution clearly is detrimental and we must look at ways to reduce pollution in the environment," said Lucking.An earlier study published in the 13th September issue of the NEJM , also by UK and Swedish researchers, showed that men with coronary heart disease who inhaled diesel fumes experienced a three fold increase in stress on the heart.
2007年11月13日星期二
2007年11月12日星期一
诡异的电影<不能说的秘密>
今天没事又看了两部电影,你现在听到的歌就是电影<十面埋妇>里的插曲,是不是有点搞笑的感觉。 《不能说的秘密》没看前，据了解是爱情片吧，前三分之二的情节也是按这个思路走的。后三分之一，难道是鬼片？人鬼情未了。除了周杰伦外，任何人都不知道女主角的存在。随剧情发展原来是科幻片，女主角穿越时空，来到20年后。期间还有一点惊险片的情节，就是学校拆毁琴房那里。 简直可以用峰回路转来形容，呵呵。
2007年11月11日星期日
Results Of Dental Health Study Of Minority New York City Youth
Results Of Dental Health Study Of Minority New York City YouthHispanic youth report better dental health habits than their non-Hispanic peers, according to a study of northern Manhattan adolescents by researchers at Columbia University Mailman School of Public Health. The study, which is published in the November issue of the Journal of Health Care for the Poor and Underserved, provides insight into the oral health of the diverse Hispanic community in America. The study, a snapshot of more than 3,200 children ages 12 to 16, who live in the northern Manhattan communities of Central Harlem and Washington Heights/Inwood, found that 94 percent of the youth responding to the study were Hispanic or black. More then 2,300 of the respondents identified themselves as Hispanic, and the greatest number of the Hispanic adolescents was of Dominican descent. In most national studies of children's oral health, the data on Hispanics largely reflects Mexican-American youth. "The study provides important information on the oral health for a Hispanic subgroup other than Mexican Americans, said Luisa N. Borrell, DDS, MPH, assistant professor of Epidemiology at the Mailman School of Public Health, and at Columbia University College of Dental Medicine, and co-author of the study. "Studies focusing on other Hispanic subgroups will help us understand the difference within the Hispanic population and underscore the need to examine health outcomes for each Hispanic subgroup whenever data is available," Dr. Borrell noted. The study relied on questionnaires filled out by the youth and clinical exams performed during each child's visit to a school-based dental clinic. Researchers found cavities in 52 percent of the Hispanic participants and 54 percent of the black youth. "This study may help us define the oral health status of Hispanic subgroups other than Mexican Americans. We still have a lot to learn about what factors are protective for the oral health of these kids, and what will work to improve that health," observed Dr. Borrell. Overall, dental health and health promoting habits of the Hispanic children were better than the other participants in the study. Ninety-four percent of Hispanic youths reported that they brush daily compared with 83 percent of blacks and 85 percent of the other children in the study. Hispanic youths were also more likely to floss. Many more Hispanic youths reported having had a dental visit sometime in their lifetime. Researchers noted moderate-to-abundant plaque in 27 percent of the Hispanic adolescents, compared with 36 percent of blacks and other children in the study. "The study's findings need to be interpreted with caution as we did not have information on the education and income of the adolescent participants' families. Also, we didn't know what proportion of these children were foreign born, which can be a protective effect for health," Dr. Borrell said.
2007年11月9日星期五
Lowering Homocysteine With B Vitamins Does Not Reduce Cardiovascular Risk
GetABI Study Finds That Even Mild Atherosclerosis In The Legs Increases Mortality SubstantiallyPatients with atherosclerosis in the leg arteries face a substantially increased all cause and cardiovascular mortality risk, according to a large study presented at the European Society of Cardiology Congress in Vienna.Heart attacks and strokes as a result of atherosclerosis have been ranked for years among the most common causes of deaths in Europe. Another previously underestimated manifestation of atherosclerosis is peripheral arterial disease (PAD), which is closely associated with heart attack or a stroke.The German epidemiological study on Ankle Brachial Index (getABI) was initiated in 2001 to answer questions about whether a simple screening test on atherosclerosis can be applied to identify it at an early stage, and if so, what risk such patients carry in the future. Professor Curt Diehm from the Clinic Karlsbad-Langensteinbach, an affiliated teaching hospital of the University in Heidelberg, and his co workers from various renowned medical institutions in Germany presented a 5 year study follow-up.Professor Diehm explained: "We used the ankle brachial-index (ABI), which is simple to understand and to apply by physicians and nurses. In an individual in the supine position, the blood pressure in the leg arteries is equal to or a little higher than in the arm arteries. If atherosclerotic stenoses in the legs manifests (termed PAD), blood flow after the obstruction decreases, and the pressure in the leg artery is lower than in the arm. This sign is almost and reliable as angiography to identify your atherosclerotic risk patient."The study included a total of 6,880 unselected patients in primary care, which underwent ABI testing by their primary care physician. Mean age of the patients was 72.5 years, 58% were females, 46% were past or current smokers, 74% had hypertension, 24% diabetes mellitus and 52% lipid disorders. Of all patients, 18.0% in the total cohort had a pathological ABI test, but the majority of these patients had no clinical signs or complaints.After a 5 year observation period, all cause mortality was 24% in patients with symptomatic PAD, 19% with asymptomatic PAD (i.e., pathological ABI but no complaints), and 9% in patients without PAD. Even when all other known risk factors for cardiovascular death were accounted for by statistical means, PAD had the best ability to predict future death, stroke or myocardial infarction.Professor Diehm said, "The bad news is: we showed that in primary care every fifth patient aged 65 years or older has atherosclerosis in the leg arteries. Because atherosclerosis is not a local process but at the same time progresses in the heart and brain vessels, such patients usually die from heart attacks or stroke. The good news is that the ABI test is not limited to expert use but can be performed in general practice. Thus, family physicians can identify high risk patients and initiate and maintain effective treatment in this large group."The study also showed that the extent of the blood pressure difference between legs and arms matters: the higher the spread between both pressures is (in other words: the lower the ABI), the higher is the mortality of patients.Professsor Diehm said that every effort should be made to implement the ABI screening in standard programs for elderly patients and patients with cardiac risk factors such as diabetes or hypertension. "A huge number of lives could be saved if patients with atherosclerosis would be identified with the ABI, and treated timely."
2007年11月6日星期二
峨眉山3日详细流水
本来是去年国庆计划去的，结果后来放弃了，这次除了张磊两口子没去，基本上都是五一去草海的原班人马，只是多了两个罗琳叫上的她的两个朋友。 成员有：周云、李姐、涵涵（8岁）、罗琳、陈婷、钟声、赖科、我。共8人。 原订计划：3号星期五坐6点10分自贡-峨眉的车出发，大概12点钟左右到峨眉，吃午饭后大概2点钟左右开始进山，从前山往上爬。晚上住宿前必须到达洪椿坪，如果时间允许可以去仙峰寺，住宿在仙峰寺是最理想的，基本可以保证第二天晚上抵达金顶，而住宿在洪椿坪第二天登顶就要吃力多了。4号星期六要到达金顶。5号一早起来看日出，在下午2点半以前坐最后一班车回自贡。计划是完美的，但是实际执行起来，就完全乱了，完全没有预计的那么容易，下面就把详细的行程整成流水帐，看看我们的旅途是如何的坎坷地，惨啊。。。。呵呵！ 2号我从下午开始收拾东西，反正想啊想的就 要装点东西进背包里，或者从里面拿出来！一直整到3号凌晨2点过才睡。我背包里的东西有：旅游鞋一双、毛绒外套一件、望远镜、薄的牛仔裤一条、体恤两件、雨伞、饭盒、筷子、牙刷、手机充电器、针线、扑克、扇子。估计重量在15斤左右，到第二天早上出发一背才发觉好重哦，但是已经来不及了清理了，估计也清理不出什么名堂，都是觉得应该带上的物品。 5：30的闹铃，然后挨个打骚扰电话，但估计他们比我还起的早。5点50坐两轮到了客运站。一看除了罗琳外，都到了。等了5分钟，罗琳到了。我、小周、罗琳三人都是背的大背包，估计重量都在15到20斤左右。爬山都背这么重，汗啊。 6：14 大巴车出站。 6：33 到达长土。 6：50 到达白房。 7：11 到达一个不晓得名字的小镇。 7：50 到达鼎新。 9：00 到荣县。自贡到荣县的路修了几年，仍然还在修，路太烂了，完全就是条4车道的机耕道。这一段路坐车很刺激，下坡的时候就象坐海盗船失重的感觉差不多。罗琳、陈婷、涵涵、李姐都有不同程度的晕车。李姐最惨，先是一个人霸占了一根板凳，睡在上面。后来来了一个婆婆要说要坐前面免得晕车，李姐只好起来，结果晕车反应就更大了，吐了。反正车上的人很多都吐了，车的后面一股怪味，幸好我坐在前面味道不是很重。车过了荣县路况好了很多，大家晕车的感觉减轻了许多。 9：45 到达长山已经到了自贡的边界了。 11：20到达乐山。这里有个地名叫沙湾。我们都说，要到自贡了，喊师傅在十字口刹一脚，呵呵。更搞笑的是，钟声不知是看到了嘉陵江还是岷江，就问卖票的大姐，这是大渡河吧，把我们笑到了住（我刚看查了下地图，好象是岷江和大渡河，汗啊）。同时司机告诉我们离峨眉只有10多分钟了，大家都期待着。。。。。。。 11：45终于抵达目的地-峨眉山长途车站。到候车室看了回自贡的车的时刻。每天直达市区的最晚一班车是下午2点半，不过有到其他市的车要走自贡过，比如到宜宾的可以在板桥高速路口下，还有到富顺的，到卢洲的可以在邓关下车等等。 12：15进入背对车站的右手边的一条岔路里的饭馆吃饭。只记得其中一个是回锅肉。吃完饭，我们又走到车站，短了两个的士，一个15元，到报国寺。在景区门口的广场照了几张相，去报国寺。结果走错了路，从一座小山上走下来，结果报国寺在山的另一边，还好路程不远，我又返回去。从报国寺左边的上山公路走到伏虎寺这段路程是1公里。路过伏虎寺没进去，因为要赶路。过了伏虎寺，路过善觉寺也没上去，因为上面是条死路，难得跑上跑下的。 13：55到了卖门票的地方，我们几个有学生证，结果都没通过验证。要求是：不能是成教的学生，所以你的学生证只要显示有成教几个字，就不行。还要求每学期都要注册盖章，不然不行。还有就是入学时间，超过了3年或者4年应该属于已经毕业的也不行。反正她们会一丝不苟的找你的证件所有的漏洞，尽最大努力让你买不到学生票。小孩子1米1以下不买票，以上买半票，1米4买全票。拿到票后，就去照相，他们会把照片直接打印到门票上面，门票是两日有效，可以进出。由于拐杖太贵了，他们一人买了一根枯竹杆，一元一根。我没买，觉得没必要。 14：40到达雷音寺，这里有很多蜜蜂乱飞。也是这一段路检测出了我们的体能。由于这一段上路比较陡，体能比较差的小周同志，已经感到力不从心了，而且还带了个8岁的孩子，加上十多斤的背包，再加这里还不算是真正的上山，万里长征第一步还没踏出来。我们当初对体能的估计过于乐观，对物品的准备也太过于细致。同志们切记啊，爬山千万别带太多东西啊，真的背起老火啊！ 15：45到达纯阳殿，好象是这一段路找到一个挑夫，因为大家的背包太重了，前面又还有很多路要走，因为计划是今天要走到洪春坪的。而有几个人的体能又支持不住，所以只好出钱叫他把我们4个人的背包挑到清音阁再说，说好是50元。 16：10到达神水阁，在这里小周、李姐、陈婷和罗琳、钟声、赖科对行走的线路产生了分歧（后面简称为1队和2队）。1队因为对自己的体能产生严重怀疑，理由如前所述：都是上山爬坡，东西又重，还有个小孩，体能根本就支持不住，速度慢了，2天根本没有从前山爬到金顶的可能。要求改变线路：马上去五显岗坐车上金顶住一晚，第二天看日出，然后走路下山。2队的意图我不清楚，但估计和我当时的想法（注意：这只是当时的想法）是一样的：既然来峨眉山，就是要爬上去，才有意义。最后2队只好把辎重扔给1队，带了些必要的东西，轻装上山。1队去五显岗坐车先上金顶。我先是打算去爬山，但是李姐劝我跟他们一路，我考虑了下，同意了（事后证明，酱紫做是完全正确地，节约了钱，沿途的重要景点也看了）。 17：15我们1队走到五显岗，结果没有上金顶的车了。而且第二天最早的车6：20发车，而金顶日出是6点10分出来，看来日出也没法看了。只好去找住宿的地方，在 峨眉山讲解投诉管理中心 旁边的小巷里有家私人旅店还不错，2人间住5个人总共才84元，还有电视、空调、单独的厕所洗澡间、女厕所有个甩干机，老板的服务态度还多好，还可以在楼下吃饭（不免费）。离五显岗车站也很近，出门不到50米远。 17：45我们收拾停当，吃了点零食，出发去清音阁游玩，路程大概是1.5公里，离的不远。路边很多买纪念品的小摊。看到有卖乌木的生肖牌，价格也不贵，结果因为不知道朋友的生肖只好作罢，打算买点蝴蝶标本做的书签带回去送，嘿嘿，价格便宜嘛。途中路过一个人工修成的大水塘，居然有一棵树长在水里，很奇特，不过没什么树叶，不晓得是不是因为被水泡了才这样地。塘边还有索道我们上去照了几张相。再望前走，看到一个景点叫 唐老鸭树，我们看了半天，都没看明白对面的树哪里象唐老鸭。有走了10多分钟，来到牛心亭下面类似一个水坝的地方，正好水坝正在放水，那里有几个学生在耍，我们也跑过去跑到坝上去走。我还跑到坝下面去淌水，水冰凉的，我下去的时候没踩斗刹车，结果把短裤都打湿了。还抓到一个小蝌蚪。这里的蝌蚪最大的有摄象头那么大，小周说是弹琴蛙的蝌蚪，有点恐怖哦。玩完水，我们来到牛心亭，照了几张相，觉得这里很凉快，我还抱着牛心石照了相。又到庙里看了看，又慢慢往回走。来到峨眉山讲解投诉管理中心，我们就进去借用电脑把小周一天照的相片打成包传到我的口口网络硬盘里，因为他的相机内存比较小。回到旅店，点了2碗豆花和一个酸菜粉丝汤，再拿出自带的抽了真空的冷吃肉吃饭，没想到饭有点馊，只好吃稀饭。自带冷吃肉很必要，尤其是上山后，可以节约点菜钱。吃着吃着还停了10分钟的电。吃完，回房间，洗澡，洗衣服，浪衣服，一天的消费情况盘存一气呵成。发短信得知2队爬到晚上9点过才找到住的地方，而且是走的后山。我们还拿出在管理中心拿的免费资料研究起路线来，发现一下午的行程都是围着报国寺绕圈，绕了一大圈没走多远，如果走五显岗直插清音阁、一线天，估计晚上可以到洪椿坪，不过也幸好转这一圈，不然一上去就没退路了，只有硬往上爬，体能影响速度，1天半是肯定无法到达金顶的。其结果是超出3天的时间才能回自贡或者是从山上退下去再想办法，那样的话行程就彻底乱了。一看时间，都11点过了，赶快开空调睡了。 〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓〓
8月4日星期六 5：20 起床，一看昨晚浪在门口的衣服，没干。OS、ON、刷牙、洗脸，整理装备，把不要的东西都寄存在旅店，我的东西基本上都要用的，所以我背上了我沉重的背包上路了。 6：05 出门。 6：10 买好了6：20发车去金顶的车票。 6：45 晚点35分钟后，中巴车终于到了。go go go .... 7：40 到达公路 零公里 车场。 8：55 车子通过剪票口，终于又上路了。因为车里大部分人都没票或者有票没去照相，所以要下车去排队买票照相才能进山。结果害我们等了一个多小时。 9：45 离雷洞坪还有一里路的地方因为堵车，只好下来步行了。 10：15经过短暂的休整，开始踏上最后的登顶行程。因为听说前面有猴子，所有暴露在外的口袋，都放进包里了。结果这里的猴子相当的规矩还有人看守。 10：55到达接引殿，考虑到后面还有6公里上山的路程，把我的包寄存在了接引殿，只带了必要的物品，提了一个塑料的袋子继续往上冲刺。这里终于体会到了爬山的痛苦，累啊，提着个口袋都很累，就不提如果是背那个背包了。 12：40到达太子坪。 13：10终于到了金顶大酒店，并和1队顺利会师，历史性的一刻。此刻大家都先到了井冈山。2队昨天和我们分手后的线路为：中峰寺-广福寺-清音阁-白龙洞-万年寺-息心所-长老坪-初殿，这么长的线路从16：30爬到21：30。第二天12：45到达金顶。金顶修建的东西太新了，没的历史沧桑的感觉。只是十面佛有点巨大，我还跑到里面去看了看。上次来，还看到了日出和贡嘎山，并且可以看到山底的风光。这次日出、云海、佛光什么都没捞到，雾气又大，山下什么都看不到，就更不提看什么贡嘎山了。不过看到周围的雾气随风而动，翻滚腾挪，还是蛮壮观的。雾气会瞬间飘来，把周围的一切都遮的严严实实，雾气转瞬过去后，一切又显现出来，而且雾气很凉爽，被太要晒久了用雾气降降温还不错。只要不在金顶住宿，根本不用带厚衣服或者租大衣，一件长袖体恤足以。 14：45坐上缆车，用了10分钟不到就到了接引殿。休息15分钟再接着走。这里我把旅游孩换了，再穿旅游鞋走下去，我的脚指头肯定着不猪，还是穿凉鞋舒服点。 15：10又要开始征程了，加油先到雷洞坪再说，只有1.5公里，又是下山，应该要不倒好多时间。 15：20我就说吧，要不了多少时间的，到了三。上去用了40分钟，下来才10分钟，不会吧。 15：35经过1队与2队的紧急磋商最后1队和2队从新组合。1队：周云、李姐、涵涵、陈婷、赖科、我，线路为雷洞坪经前山走下山去；2队：罗琳、钟声，因爬山上来，体能消耗过大，无法再继续走下山去，所以坐车下去再找地方住宿。一出发，陈婷和赖科就冲到前面去了，毕竟是年轻人啊，有活力，再加上他们星期一要赶回去上班不能耽搁，最迟必须坐明天下午2点半的最后一班车回自贡，不然打地都要回去。剩下我、小周、李姐、涵涵慢慢往下撤，本来小周是计划他和涵涵先撤下山的，但是李姐认为时间够用，坚持要他们一家人都走下山去，免得留下遗憾。小周一路上都在说，是被李姐诓来的，他和涵涵的体能哪里能爬山嘛。特别是涵涵一个8岁的小朋友，头天走了起码有10多公里路，早上又从雷洞坪爬上金顶，又是个9公里。对一个8岁的小孩子来说，真是不容易啊，而且一路上经管脚痛，但是没有主动的叫她爹妈背过，都是她爹地主动的去背她，她还不情愿。也没吵着要吃这样吃那样。浪乖的娃儿很少见哈。也幸好她这么乖，不然后面的路可咋走哦。估计她妈老汉弄她来爬山，就是知道她不会耍赖不走的。 17：00越往下走，雾气越大。我们一瘸一拐的下到洗象池，这里正在维修，洗象池的是水也脏的不得了，不过这里有很多猴子，幸好这里的猴子还比较文明，看来是学习过三个代表，是一个合格的和谐猴子。我们没敢招惹他们，他们也没理我们，就这样走过去了。而且我们的饼干和火腿肠就提在手里，它们也没抢，这一点迷惑了我们，以为峨眉山的猴子都是和谐猴，为后面的危险埋下了伏笔。 17：15我们在洗象池照了几张相后，继续往下赶路。 18：20走下钻天坡，来到九岭岗。从钻天坡上已经可以望见遇先寺了。但那里还不是我们的目的地，主要是那里没有住宿的地方。这个钻天坡真是陡啊，几乎是40度的梯子扶摇直上，幸好我们是往下走，不然要耗费很大的体能才能上去。现在回想起来，估计这里算是整个峨眉山最险要也最考验体能的地段，99道拐根本不算什么。在坡上我们遇到几个虔诚的朝圣者，三步一扣首，一步三个台阶。九领岗是前山和后山步行道的交汇合点。过了九岭岗，又是一个下山的道路，在路过一个路边店时，我们发现店子的前面有只猴子，李姐赶快拿出饼干来喂，没想到店子的下面路上还有两只猴子，这两只猴子一看就是路霸，因为一只老青猴就直接坐在路的中央，一副收过路费的样子。我们想把装饼干和火腿肠的袋子藏起来，已经来不及了。李姐抓了一把饼干，扔到路边，乘两只猴子争抢时，溜了过去。但类2个猴子明显是长期在这一带打劫路人的专业户，职业素质那是相当的高，把饼干塞到嘴里装起，又来抢李姐手里的饼干。它们龇牙咧嘴的，嘴里还怪叫，李姐怕它们扑到她身上来抢，吓的她尖叫一声把手的口袋和拐杖就扔地上了。猴子拖着袋子往旁边的悬崖跑去，幸好我的一双旅游鞋没和装食品的袋子拴一起，不然也一并拖去了，呵呵。反正就是吃的东西一定要放在背包里，遇到这种半路杀出来的猴子，就要小心了。或者准备点饼干装成小袋，递给它们当过路费，估计它们看你手里没东西了，就会放行的。 18：50到达遇仙寺。这里我在一个宜宾人发在论坛里的照片看到过，因为这里在搞维修，有一个小的搅拌机，他奇怪的是遇仙寺在山的半中腰，两边都是台阶路，这个搅拌机是怎么运到这里的。我也纳闷，所以问了那里的民工，原来是先把搅拌机通过公路运到五显岗，再从洗象池、九岭岗人工抬到这里的，无法想象他们当时是怎么抬到这里的，修缮用的石子是用附近的石头在现场打的。收到赖科发的消息，说他们两人已经到了仙峰寺，并且已经订了房间。 19：10下到遇仙寺所在山谷的谷底，这里有个路边店，我们点了一个汤，一份素菜，然后拿出冷吃肉开始用餐，因为天色越来越暗，怕前面没吃的地方了，还有就是也实在饿了，吃的东西又被猴子抢了。现在冷吃肉的作用发挥出来了，吃了13块钱，还吃的比较满意。 19：35出发往今晚的住宿地仙峰寺进发。听店老板说，还有半个小时的路程就到仙峰寺，我们想他们要半个小时，我们应该一个小时可以到，毕竟我们的速度要慢点，这样算来8点半左右可以到，因为一般是9点钟完全天黑。不过经过一天的跋涉，涵涵的脚已经受不了了，一直嚷脚痛，但是仍然坚持着和大人走。走到离仙峰寺还有大概一公里路时，居然哭了，毕竟走了一天，而且天色又晚了，对小孩子来说，基本达到了体能的极限了。正好这时抬滑竿的师傅下班回家，路过，就讲好价钱，一个小孩20元，抬到仙峰寺，先涵涵还不愿意，说要自己走，我们古斗她坐上去，这也是她全程唯一一次坐滑竿。我把背包也给滑竿师傅背了，大家都轻装继续最后的冲刺，在滑竿的带领下，小周的速度也提了起来，很快就到了仙峰寺，发了消息叫赖科和陈婷出来接我们，幸好他们抢先到达订好了房间，不然我们后来的话，就只能分开到各个房间去住了，而不能住在一间房间里面。6个人住了个7人间175元。赖科和陈婷他们冲到洗象池子，本来说要等我们的，但是看到很多人都往仙峰寺赶，他们也慌了，怕到时没的住处，连忙星也往仙峰寺冲，路上还听说有猴子拦路抢劫，劫食不劫色，已经有十多个人被抢了，陈婷吓惨了，怕被猴子抢去当压寨猴子，帽子也不敢戴了，拼命抓住赖科的胳膊才冲过那一段猴子出没的地带。休息了一会儿，我们听说仙峰寺有洗脚房，于是周云、赖科、我便下楼一路寻去，终于在女厕所旁边找到了洗脚房，这个洗脚房居然还有提供免费洗浴中心，只不过当我们进去刚说脱衣服时，才知道这里只提供冷水。因为仙峰寺地段的气候比较低大概22度左右，我们没敢洗冷水。出来到大厅的洗脚房，找了两个盆子，一人放了一盆热水泡脚，走了一天，脚都要冒烟了，当时的感觉就是人生最大的幸福莫过于洗脚（洗脚、洗澡免费）。洗完脚，又是商量明天的线路，及一天消费的盘存。商量的结果为：陈婷、赖科因为后天星期一要上班，最晚必须坐明天下午2点半的最后一班车回自贡。所以明天中午以前他们要冲下山去，不能等我们一路。而剩下的人，小周虽然星期一也要上班，但是体能有限，对剩下的路程估计半天是走不下去了，只能走多少算多少，尽力往下赶，预计是下午3点左右才能下山，但已经无法坐到2点半的车了，所以只能在山下住一晚，星期一早上再坐车回自贡。我由于星期二才上班，所以可以跟小周一家人一起慢慢往下走。一看时间10点过了累了一整天，赶快睡了。
8月4日星期六 5：20 起床，一看昨晚浪在门口的衣服，没干。OS、ON、刷牙、洗脸，整理装备，把不要的东西都寄存在旅店，我的东西基本上都要用的，所以我背上了我沉重的背包上路了。 6：05 出门。 6：10 买好了6：20发车去金顶的车票。 6：45 晚点35分钟后，中巴车终于到了。go go go .... 7：40 到达公路 零公里 车场。 8：55 车子通过剪票口，终于又上路了。因为车里大部分人都没票或者有票没去照相，所以要下车去排队买票照相才能进山。结果害我们等了一个多小时。 9：45 离雷洞坪还有一里路的地方因为堵车，只好下来步行了。 10：15经过短暂的休整，开始踏上最后的登顶行程。因为听说前面有猴子，所有暴露在外的口袋，都放进包里了。结果这里的猴子相当的规矩还有人看守。 10：55到达接引殿，考虑到后面还有6公里上山的路程，把我的包寄存在了接引殿，只带了必要的物品，提了一个塑料的袋子继续往上冲刺。这里终于体会到了爬山的痛苦，累啊，提着个口袋都很累，就不提如果是背那个背包了。 12：40到达太子坪。 13：10终于到了金顶大酒店，并和1队顺利会师，历史性的一刻。此刻大家都先到了井冈山。2队昨天和我们分手后的线路为：中峰寺-广福寺-清音阁-白龙洞-万年寺-息心所-长老坪-初殿，这么长的线路从16：30爬到21：30。第二天12：45到达金顶。金顶修建的东西太新了，没的历史沧桑的感觉。只是十面佛有点巨大，我还跑到里面去看了看。上次来，还看到了日出和贡嘎山，并且可以看到山底的风光。这次日出、云海、佛光什么都没捞到，雾气又大，山下什么都看不到，就更不提看什么贡嘎山了。不过看到周围的雾气随风而动，翻滚腾挪，还是蛮壮观的。雾气会瞬间飘来，把周围的一切都遮的严严实实，雾气转瞬过去后，一切又显现出来，而且雾气很凉爽，被太要晒久了用雾气降降温还不错。只要不在金顶住宿，根本不用带厚衣服或者租大衣，一件长袖体恤足以。 14：45坐上缆车，用了10分钟不到就到了接引殿。休息15分钟再接着走。这里我把旅游孩换了，再穿旅游鞋走下去，我的脚指头肯定着不猪，还是穿凉鞋舒服点。 15：10又要开始征程了，加油先到雷洞坪再说，只有1.5公里，又是下山，应该要不倒好多时间。 15：20我就说吧，要不了多少时间的，到了三。上去用了40分钟，下来才10分钟，不会吧。 15：35经过1队与2队的紧急磋商最后1队和2队从新组合。1队：周云、李姐、涵涵、陈婷、赖科、我，线路为雷洞坪经前山走下山去；2队：罗琳、钟声，因爬山上来，体能消耗过大，无法再继续走下山去，所以坐车下去再找地方住宿。一出发，陈婷和赖科就冲到前面去了，毕竟是年轻人啊，有活力，再加上他们星期一要赶回去上班不能耽搁，最迟必须坐明天下午2点半的最后一班车回自贡，不然打地都要回去。剩下我、小周、李姐、涵涵慢慢往下撤，本来小周是计划他和涵涵先撤下山的，但是李姐认为时间够用，坚持要他们一家人都走下山去，免得留下遗憾。小周一路上都在说，是被李姐诓来的，他和涵涵的体能哪里能爬山嘛。特别是涵涵一个8岁的小朋友，头天走了起码有10多公里路，早上又从雷洞坪爬上金顶，又是个9公里。对一个8岁的小孩子来说，真是不容易啊，而且一路上经管脚痛，但是没有主动的叫她爹妈背过，都是她爹地主动的去背她，她还不情愿。也没吵着要吃这样吃那样。浪乖的娃儿很少见哈。也幸好她这么乖，不然后面的路可咋走哦。估计她妈老汉弄她来爬山，就是知道她不会耍赖不走的。 17：00越往下走，雾气越大。我们一瘸一拐的下到洗象池，这里正在维修，洗象池的是水也脏的不得了，不过这里有很多猴子，幸好这里的猴子还比较文明，看来是学习过三个代表，是一个合格的和谐猴子。我们没敢招惹他们，他们也没理我们，就这样走过去了。而且我们的饼干和火腿肠就提在手里，它们也没抢，这一点迷惑了我们，以为峨眉山的猴子都是和谐猴，为后面的危险埋下了伏笔。 17：15我们在洗象池照了几张相后，继续往下赶路。 18：20走下钻天坡，来到九岭岗。从钻天坡上已经可以望见遇先寺了。但那里还不是我们的目的地，主要是那里没有住宿的地方。这个钻天坡真是陡啊，几乎是40度的梯子扶摇直上，幸好我们是往下走，不然要耗费很大的体能才能上去。现在回想起来，估计这里算是整个峨眉山最险要也最考验体能的地段，99道拐根本不算什么。在坡上我们遇到几个虔诚的朝圣者，三步一扣首，一步三个台阶。九领岗是前山和后山步行道的交汇合点。过了九岭岗，又是一个下山的道路，在路过一个路边店时，我们发现店子的前面有只猴子，李姐赶快拿出饼干来喂，没想到店子的下面路上还有两只猴子，这两只猴子一看就是路霸，因为一只老青猴就直接坐在路的中央，一副收过路费的样子。我们想把装饼干和火腿肠的袋子藏起来，已经来不及了。李姐抓了一把饼干，扔到路边，乘两只猴子争抢时，溜了过去。但类2个猴子明显是长期在这一带打劫路人的专业户，职业素质那是相当的高，把饼干塞到嘴里装起，又来抢李姐手里的饼干。它们龇牙咧嘴的，嘴里还怪叫，李姐怕它们扑到她身上来抢，吓的她尖叫一声把手的口袋和拐杖就扔地上了。猴子拖着袋子往旁边的悬崖跑去，幸好我的一双旅游鞋没和装食品的袋子拴一起，不然也一并拖去了，呵呵。反正就是吃的东西一定要放在背包里，遇到这种半路杀出来的猴子，就要小心了。或者准备点饼干装成小袋，递给它们当过路费，估计它们看你手里没东西了，就会放行的。 18：50到达遇仙寺。这里我在一个宜宾人发在论坛里的照片看到过，因为这里在搞维修，有一个小的搅拌机，他奇怪的是遇仙寺在山的半中腰，两边都是台阶路，这个搅拌机是怎么运到这里的。我也纳闷，所以问了那里的民工，原来是先把搅拌机通过公路运到五显岗，再从洗象池、九岭岗人工抬到这里的，无法想象他们当时是怎么抬到这里的，修缮用的石子是用附近的石头在现场打的。收到赖科发的消息，说他们两人已经到了仙峰寺，并且已经订了房间。 19：10下到遇仙寺所在山谷的谷底，这里有个路边店，我们点了一个汤，一份素菜，然后拿出冷吃肉开始用餐，因为天色越来越暗，怕前面没吃的地方了，还有就是也实在饿了，吃的东西又被猴子抢了。现在冷吃肉的作用发挥出来了，吃了13块钱，还吃的比较满意。 19：35出发往今晚的住宿地仙峰寺进发。听店老板说，还有半个小时的路程就到仙峰寺，我们想他们要半个小时，我们应该一个小时可以到，毕竟我们的速度要慢点，这样算来8点半左右可以到，因为一般是9点钟完全天黑。不过经过一天的跋涉，涵涵的脚已经受不了了，一直嚷脚痛，但是仍然坚持着和大人走。走到离仙峰寺还有大概一公里路时，居然哭了，毕竟走了一天，而且天色又晚了，对小孩子来说，基本达到了体能的极限了。正好这时抬滑竿的师傅下班回家，路过，就讲好价钱，一个小孩20元，抬到仙峰寺，先涵涵还不愿意，说要自己走，我们古斗她坐上去，这也是她全程唯一一次坐滑竿。我把背包也给滑竿师傅背了，大家都轻装继续最后的冲刺，在滑竿的带领下，小周的速度也提了起来，很快就到了仙峰寺，发了消息叫赖科和陈婷出来接我们，幸好他们抢先到达订好了房间，不然我们后来的话，就只能分开到各个房间去住了，而不能住在一间房间里面。6个人住了个7人间175元。赖科和陈婷他们冲到洗象池子，本来说要等我们的，但是看到很多人都往仙峰寺赶，他们也慌了，怕到时没的住处，连忙星也往仙峰寺冲，路上还听说有猴子拦路抢劫，劫食不劫色，已经有十多个人被抢了，陈婷吓惨了，怕被猴子抢去当压寨猴子，帽子也不敢戴了，拼命抓住赖科的胳膊才冲过那一段猴子出没的地带。休息了一会儿，我们听说仙峰寺有洗脚房，于是周云、赖科、我便下楼一路寻去，终于在女厕所旁边找到了洗脚房，这个洗脚房居然还有提供免费洗浴中心，只不过当我们进去刚说脱衣服时，才知道这里只提供冷水。因为仙峰寺地段的气候比较低大概22度左右，我们没敢洗冷水。出来到大厅的洗脚房，找了两个盆子，一人放了一盆热水泡脚，走了一天，脚都要冒烟了，当时的感觉就是人生最大的幸福莫过于洗脚（洗脚、洗澡免费）。洗完脚，又是商量明天的线路，及一天消费的盘存。商量的结果为：陈婷、赖科因为后天星期一要上班，最晚必须坐明天下午2点半的最后一班车回自贡。所以明天中午以前他们要冲下山去，不能等我们一路。而剩下的人，小周虽然星期一也要上班，但是体能有限，对剩下的路程估计半天是走不下去了，只能走多少算多少，尽力往下赶，预计是下午3点左右才能下山，但已经无法坐到2点半的车了，所以只能在山下住一晚，星期一早上再坐车回自贡。我由于星期二才上班，所以可以跟小周一家人一起慢慢往下走。一看时间10点过了累了一整天，赶快睡了。
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